@article {27087,
	title = {Computational quantification of patient-specific changes in ventricular dynamics associated with pulmonary hypertension},
	journal = {American Journal of Physiology-Heart and Circulatory Physiology},
	volume = {31711911},
	year = {2019},
	pages = {H1363 - H1375},
	publisher = {American Journal of Physiology},
	author = {Finsberg, Henrik and Xi, Ce and Zhao, Xiaodan and Le Tan, Ju and Genet, Martin and Sundnes, Joakim and Lik Chuan {Lee} and Zhong, Liang and Wall, Samuel}
}
@misc {26384,
	title = {Adjoint Based Personalization of Mechanical Models for Quantification of Right Ventricular Failure in Pulmonary Hypertension},
	year = {2018},
	address = {Heart by Numbers Conference, Berlin, Germany},
	author = {Finsberg, Henrik and Xi, Ce and Xiaodan, Zhao and Le Tan, Ju and Genet, Martin and Sundnes, Joakim and Lik Chuan {Lee} and Zhong, Liang and Wall, Samuel}
}
@article {25636,
	title = {Efficient estimation of personalized biventricular mechanical function employing gradient-based optimization},
	journal = { International Journal for Numerical Methods in Biomedical Engineering},
	volume = {34},
	year = {2018},
	publisher = {John Wiley \& Sons},
	doi = {10.1002/cnm.2982},
	author = {Finsberg, Henrik and Xi, Ce and Le Tan, Ju and Zhong, L and Genet, Martin and Sundnes, Joakim and Lik Chuan {Lee} and Wall, Samuel}
}
@article {25483,
	title = {An integrated electromechanical-growth heart model for simulating cardiac therapies},
	journal = {Biomechanics and modeling in mechanobiology},
	volume = {15},
	number = {4},
	year = {2016},
	pages = {791{\textendash}803},
	publisher = {{Springer Berlin Heidelberg},
	abstract = {An emerging class of models has been developed in recent years to predict cardiac growth and remodeling. We recently developed a cardiac  constitutive model that predicts remodeling in response to elevated hemodynamics loading, and a subsequent reversal of the remodeling process when the loading is reduced. Here, we describe the integration of this model to an existing strongly coupled electromechanical model of the heart. },
	author = {Lik Chuan {Lee} and Sundnes, Joakim and Genet, Martin and Jonathan F {Wenk} and Wall, Samuel}
}
@article {24650,
	title = {Physics-based computer simulation of the long-term effects of cardiac regenerative therapies},
	journal = {Technology},
	volume = {4},
	number = {01},
	year = {2016},
	pages = {23{\textendash}29},
	publisher = {{World Scientific Publishing Co./Imperial College Press},
	author = {Lik Chuan {Lee} and Sundnes, Joakim and Genet, Martin and Wall, Samuel}
}
@article {23708,
	title = {An integrated electromechanical-growth heart model for simulating cardiac therapies},
	journal = {Biomechanics and Modeling in Mechanobiology},
	volume = {15},
	year = {2015},
	pages = {791-803},
	publisher = {Springer Berlin Heidelberg},
	author = {Lik Chuan {Lee} and Sundnes, Joakim and Genet, Martin and Jonathan F {Wenk} and Wall, Samuel}
}
@article {Lee20143115,
	title = {Bioinjection treatment: Effects of post-injection residual stress on left ventricular wall stress},
	journal = {Journal of Biomechanics},
	volume = {47},
	year = {2014},
	pages = {3115 - 3119},
	publisher = {Elsevier},
	abstract = {Injection of biomaterials into diseased myocardium has been associated with decreased myofiber stress, restored left ventricular (LV) geometry and improved \{LV\} function. However, its exact mechanism(s) of action remained unclear. In this work, we present the first patient-specific computational model of biomaterial injection that accounts for the possibility of residual strain and stress introduced by this treatment. We show that the presence of residual stress can create more heterogeneous regional myofiber stress and strain fields. Our simulation results show that the treatment generates low stress and stretch areas between injection sites, and high stress and stretch areas between the injections and both the endocardium and epicardium. Globally, these local changes are translated into an increase in average myofiber stress and its standard deviation (from 6.9{\textpm}4.6 to 11.2{\textpm}48.8 kPa and 30{\textpm}15 to 35.1{\textpm}50.9 kPa at end-diastole and end-systole, respectively). We also show that the myofiber stress field is sensitive to the void-to-size ratio. For a constant void size, the myofiber stress field became less heterogeneous with decreasing injection volume. These results suggest that the residual stress and strain possibly generated by biomaterial injection treatment can have large effects on the regional myocardial stress and strain fields, which may be important in the remodeling process.},
	doi = {http://dx.doi.org/10.1016/j.jbiomech.2014.06.026},
	url = {http://www.sciencedirect.com/science/article/pii/S0021929014003650},
	author = {Lik Chuan {Lee} and Wall, Samuel and Genet, Martin and Hinson, Andy and Julius M. {Guccione}}
}
