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Filters: 1 is biblio_type:Poster and Author is Namit Gaur  [Reset Search]
2015
N. Gaur, X. Cai and Y. Rudy. Dysfunctional Sarcoplasmic Reticulum Ca2+ Release Underlies Arrhythmogenic Triggers in Catecholaminergic Polymorphic Ventricular Tachycardia: A Simulation Study in a Human Ventricular Myocyte Model In Gordons Research Conference on Cardiac Arrhythmia. Lucca, Italy: Gordons Research Conference on Cardiac Arrhythmia, 2015.File cpvt_poster_grc_2015.pptx (1.48 MB)
2014
N. Gaur, X. Cai and Y. Rudy. Arrhythmogenic Mechanisms and Therapeutic Targets for Catecholaminergic Polymorphic Ventricular Tachycardia: A Simulation Study in a Human Ventricular Myocyte In Simula Research Laboratory., 2014.
N. Gaur, X. Cai and Y. Rudy. Cellular Arrhythmogenesis in CPVT in a computational model of cardiac ventricular myocyte. Maastrich, Netherlands: European Working Group of Cardiac Cellular Electrophysiology, 2014.
N. Gaur, X. Cai and Y. Rudy. Cellular Arrhythmogenesis in CPVT in a Computational Model of Cardiac Ventricular Myocyte. Scandinavian Physiological Society Meeting, 2014.
N. Gaur, X. Cai and Y. Rudy. Mathematical Modeling of Ca Handling and Computational Studies of Ca-related Arrhythmogenesis in Heart In National University of Defense Technology, China. Changsha, China, 2014.
N. Gaur, X. Cai and Y. Rudy. Spontaneous Ca2+ Release and Ca2+ Waves Underlie Early and Delayed Afterdepolarizations, and Triggered Activity, in Ryanodine Receptor Mutations Associated With Catecholaminergic Polymorphic Ventricular Tachycardia., 2014.
N. Gaur, X. Cai and Y. Rudy. Spontaneous Ca2+ Release and Ca2+ Waves Underlie Early and Delayed Afterdepolarizations, and Triggered Activity in Ryanodine Receptor Mutation associated with Catecholaminergic Polymorphic Ventricular Tachycardia. Scandinavian Physiological Society, 2014.