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Filters: 1 is biblio_type:Technical reports and Author is Namit Gaur  [Reset Search]
2015
N. Gaur, X. Cai and Y. Rudy. Dysfunctional Sarcoplasmic Reticulum Ca2+ Release Underlies Arrhythmogenic Triggers in Catecholaminergic Polymorphic Ventricular Tachycardia: A Simulation Study in a Human Ventricular Myocyte Model In Gordons Research Conference on Cardiac Arrhythmia. Lucca, Italy: Gordons Research Conference on Cardiac Arrhythmia, 2015.File cpvt_poster_grc_2015.pptx (1.48 MB)
2014
N. Gaur, X. Cai and Y. Rudy. Arrhythmogenic Mechanisms and Therapeutic Targets for Catecholaminergic Polymorphic Ventricular Tachycardia: A Simulation Study in a Human Ventricular Myocyte In Simula Research Laboratory., 2014.
N. Gaur, X. Cai and Y. Rudy. Cellular Arrhythmogenesis in CPVT in a Computational Model of Cardiac Ventricular Myocyte. Scandinavian Physiological Society Meeting, 2014.
N. Gaur, X. Cai and Y. Rudy. Cellular Arrhythmogenesis in CPVT in a computational model of cardiac ventricular myocyte. Maastrich, Netherlands: European Working Group of Cardiac Cellular Electrophysiology, 2014.
N. Gaur, X. Cai and Y. Rudy. Mathematical Modeling of Ca Handling and Computational Studies of Ca-related Arrhythmogenesis in Heart In National University of Defense Technology, China. Changsha, China, 2014.
N. Gaur, X. Cai and Y. Rudy. Spontaneous Ca2+ Release and Ca2+ Waves Underlie Early and Delayed Afterdepolarizations, and Triggered Activity in Ryanodine Receptor Mutation associated with Catecholaminergic Polymorphic Ventricular Tachycardia. Scandinavian Physiological Society, 2014.
N. Gaur, X. Cai and Y. Rudy. Spontaneous Ca2+ Release and Ca2+ Waves Underlie Early and Delayed Afterdepolarizations, and Triggered Activity, in Ryanodine Receptor Mutations Associated With Catecholaminergic Polymorphic Ventricular Tachycardia., 2014.