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S. Kallhovd, S. U. Gjerald, S. Wall, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltratio is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In 2015 E-Health and Bioengineering Conference (EHB). ACM IEEE, 2015.
Proceedings, refereed
2015
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S. Kallhovd, S. U. Gjerald, O. Maennel, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltration is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In E-Health and Bioengineering Conference (EHB). IEEE, 2015.
Proceedings, refereed
2015
View
J. Koivumäki, G. Seemann and M. Maleckar

Divergent Action Potential Morphology in Human Atrial Cells Vs. Tissue: Underlying Ionic Mechanisms

In Computing in Cardiology. Vol. 39. Alan Murray, 2012.
Proceedings, refereed
2012
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A. Tveito, G. T. Lines and M. Maleckar

A Note on a Possible Pro-Arrhythmic Property of Anti-Arrhythmic Drugs Aimed at Improving Gap-Junction Coupling

Biophysical Journal 102 (2012): 231-237.
Journal Article
2012
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Export 13 results:
2015

Proceedings, refereed

S. Kallhovd, S. U. Gjerald, O. Maennel, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltration is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In E-Health and Bioengineering Conference (EHB). IEEE, 2015.
Status: Published
S. Kallhovd, S. U. Gjerald, S. Wall, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltratio is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In 2015 E-Health and Bioengineering Conference (EHB). ACM IEEE, 2015.
Status: Published

Talks, invited

M. Maleckar

How many ionic models do we need for modelling of the atria?

In Atrial Signals 2015. Karlsruhe, Germany, 2015.
Status: Published
M. Maleckar

Putting the pieces together: towards supplementing sparse clinical data with multi physics simulation

In Foundation Teofilo Rossi di Montelera Forum 2015. Lugano, Switzerland, 2015.
Status: Published
2012

Journal articles

R. A. Rose, D. Belke, M. Maleckar and W. R. Giles

Ca(2+) Entry Through TRP-C Channels Regulates Fibroblast Biology in Chronic Atrial Fibrillation

Circulation 126 (2012): 2039-2041.
Status: Published
A. Tveito, G. T. Lines and M. Maleckar

A Note on a Possible Pro-Arrhythmic Property of Anti-Arrhythmic Drugs Aimed at Improving Gap-Junction Coupling

Biophysical Journal 102 (2012): 231-237.
Status: Published
A. Tveito, G. T. Lines, M. E. Rognes and M. Maleckar

An Analysis of the Shock Strength Needed to Achieve Defibrillation in a Simplified Mathematical Model of Cardiac Tissue

International Journal of Numerical Analysis and Modeling 9 (2012): 644-657.
Status: Published
A. Tveito, G. T. Lines, A. G. Edwards, M. Maleckar, A. P. Michailova, J. E. Hake and A. D. McCulloch

Slow Calcium-Depolarization-Calcium Waves May Initiate Fast Local Depolarization Waves in Ventricular Tissue

Progress in Biophysics and Molecular Biology 110 (2012): 295-304.
Status: Published

Proceedings, refereed

J. Koivumäki, G. Seemann and M. Maleckar

Divergent Action Potential Morphology in Human Atrial Cells Vs. Tissue: Underlying Ionic Mechanisms

In Computing in Cardiology. Vol. 39. Alan Murray, 2012.
Status: Published

Posters

J. Koivumäki, G. Seemann and M. Maleckar

Divergent Action Potential Morphology in Human Atrial Cells Vs. Tissue: Underlying Ionic Mechanisms

2012.
Status: Published