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S. Kallhovd, S. U. Gjerald, S. Wall, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltratio is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In 2015 E-Health and Bioengineering Conference (EHB). ACM IEEE, 2015.
Proceedings, refereed
2015
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S. Kallhovd, S. U. Gjerald, O. Maennel, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltration is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In E-Health and Bioengineering Conference (EHB). IEEE, 2015.
Proceedings, refereed
2015
View
J. Koivumäki, G. Seemann, M. Maleckar and P. Tavi

In Silico Screening of the Key Cellular Remodeling Targets in Chronic Atrial Fibrillation

PLoS Computational Biology 10(5) (2014).
Journal Article
2014
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L. Yuan, J. Koivumäki, B. Liang, L. G. Lorentzen, J. H. Svendsen, J. Tfelt-Hansen, M. Maleckar, N. Schmitt, M. S. Olesen and T. Jespersen

Investigations of the NaV\beta1b Sodium Channel Subunit in Human Ventricle; Functional Characterization of the H162P Brugada Syndrome Mutant

Heart and Circulatory Physiology 306 (2014): H1204-H1212.
Journal Article
2014
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2015

Proceedings, refereed

S. Kallhovd, S. U. Gjerald, O. Maennel, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltration is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In E-Health and Bioengineering Conference (EHB). IEEE, 2015.
Status: Published
S. Kallhovd, S. U. Gjerald, S. Wall, J. Saberniak, K. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltratio is the primary factor determining conduction disturbance in a computational model of arrhythmogenic cardiomyopathy

In 2015 E-Health and Bioengineering Conference (EHB). ACM IEEE, 2015.
Status: Published

Talks, invited

M. Maleckar

How many ionic models do we need for modelling of the atria?

In Atrial Signals 2015. Karlsruhe, Germany, 2015.
Status: Published
M. Maleckar

Putting the pieces together: towards supplementing sparse clinical data with multi physics simulation

In Foundation Teofilo Rossi di Montelera Forum 2015. Lugano, Switzerland, 2015.
Status: Published
2014

Journal articles

M. Frisk, J. Koivumäki, P. A. Norseng, M. Maleckar, O. M. Sejersted and W. E. Louch

Variable T-Tubule Organization and Ca2+ Homeostasis Across the Atria

Heart and Circulatory Physiology 307 (2014): H609-H620.
Status: Published
J. Koivumäki, G. Seemann, M. Maleckar and P. Tavi

In Silico Screening of the Key Cellular Remodeling Targets in Chronic Atrial Fibrillation

PLoS Computational Biology 10(5) (2014).
Status: Published
J. Koivumäki, R. Clark, D. Belke, C. Kondo, P. Fedak, M. Maleckar and W. R. Giles

Na+ Current Expression in Human Atrial Myofibroblasts: Its Identity and Functional Consequences

Frontiers in Physiology 5 (2014): 275.
Status: Published
M. Maleckar, G. T. Lines, J. Koivumäki, J. M. Cordeiro and K. Calloe

NS5806 Partially Restores Action Potential Duration But Fails to Ameliorate Calcium Transient Dysfunction in a Computational Model of Canine Heart Failure

Europace 16 (2014): iv46-iv55.
Status: Published
L. Yuan, J. Koivumäki, B. Liang, L. G. Lorentzen, J. H. Svendsen, J. Tfelt-Hansen, M. Maleckar, N. Schmitt, M. S. Olesen and T. Jespersen

Investigations of the NaV\beta1b Sodium Channel Subunit in Human Ventricle; Functional Characterization of the H162P Brugada Syndrome Mutant

Heart and Circulatory Physiology 306 (2014): H1204-H1212.
Status: Published

Posters

S. Kallhovd, V. Mezzano, S. U. Gjerald, J. Saberniak, F. Sheikh, H. Haugaa and M. Maleckar

Localization and not extent of fibrofatty infiltration is the primary factor determining conduction disturbance in a computational model of arrhythomogenic cardiomyopathy

In Biophysical Society 58th Annual Meeting. Biophysical Society 58th Annual Meeting, San Francisco, February 15-19, 2014, 2014.
Status: Published